Genki Hayashi

Genki Hayashi has a diverse work experience spanning various research and clinical roles. Genki began their career as a Research Assistant Intern at the University of California, San Diego in 2007. In 2008, they worked as a Clinical Intern at the UCSF Medical Center and concurrently served as an Undergraduate Research Assistant at UC Irvine, where they analyzed the effects of Ant1 knockout on expression patterns in mouse cardiac tissue.

In 2010, Genki joined UCSF as a Research Assistant, where they contributed to the identification of genetic associations and mutations linked to psoriasis. Genki also participated in a project that investigated the protective effect of HLA alleles in psoriasis patients against HIV-1 disease.

From 2011 to 2016, Genki served as a Graduate Student at UC Davis and discovered a mouse model for Friedreich's ataxia. Genki explored the role of oxidative stress response genes and developed mRNA biomarkers in a cell model of the disease.

Since 2016, Genki has been associated with UCSF as a Postdoctoral Fellow. Genki'swork focused on understanding COL4A1-mediated intracerebral hemorrhage and the efficacy of chemical chaperones in minimizing stroke severity. Genki also investigated the role of altered TGFβ signaling in causing vascular defects associated with COL4A1.

In 2021, Genki joined Neuron23 as a Scientist, and later advanced to the role of Scientist II in 2023. Further details about their current position are not provided.

Genki Hayashi completed a Bachelor's Degree in Biology at UC Irvine from 2005 to 2009. Following this, they pursued a Doctor of Philosophy (Ph.D.) in Genetics at the University of California, Davis, from 2011 to 2016.